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Wednesday, January 30, 2013

This Man is a Korean guy named Heo Gyungyoung

Don't they look similar? What Do you guys think? Heo Gyungyoung is a famous Korean scammer who claims He can do everything like God, so I think He could be the this man. I thought this man could be a Southern European because he looks like one but now I think He could be Heo Gyungyoung!

나로호를 뛰어넘는 축구의 위력ㄷㄷㄷㄷ 나로호 교신 성공 후에도 엘클라시코가 네이버 검색어 1위임, 나로호가 2위

Monday, January 28, 2013

arab turkish greek all we are same to music comment

arab turkish greek all we are same to music. 

Romans were Mediterranean comment

brother_in_magic brother_...
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The Romans (those of Mediterranean ancestry...actually you could be a Roman and not Italian!) averaged about 5ft 6, a few inches shorter than today. Germanics were generally a few inches bigger than the average Roman, between 5ft 8 and 6 foot. It is a myth that every one in ancient times was short, most were only fractionally smaller than people now and many were just as tall, there were just less really big people on average. It was in the middle ages that people got smaller, due to overcrowded towns and poorer diets.

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    Thanks that was very informative! :D;_ylt=Ao8eRhHOU5Ok867jo8_mA0Dsy6IX;_ylv=3?qid=20121011152055AANpMLr

Sunday, January 27, 2013

How does a lack of sun affect us?

Sunshine affects the brain via the interaction of the chemicals melatonin and serotonin, as well as vitamin D. When sunlight hits your eyes, your optic nerve sends a message to the gland in the brain that produces melatonin (a hormone that helps you sleep); the gland decreases its secretions of melatonin until the sun goes down again. The opposite happens with the chemical serotonin; when you're exposed to sun, your brain increases serotonin (a hormone connected with feelings of happiness and wakefulness) production. And when the ultraviolet rays from sunshine touch your skin, your body produces vitamin D, which helps you maintain serotonin levels. Generally, we're asleep or feeling slowed down during the dark hours, and physically and emotionally up during the day. This is the human circadian rhythm. We are able to function against these biological rhythms when we must (as night-shift workers do), but it can be hard on our bodies and minds. When we go without sunshine, we can even get seasonal affective disorder (SAD); people who suffer from this disorder get depressed during the times when there's not much sun, although they're typically fine in the warmer, sunnier months of the year. SAD is most prevalent in places where there are scant sunlit hours in the winter (such as Alaska) or where it's overcast for extended periods (parts of the U.S. Northwest). SAD can often be treated with phototherapy that exposes the patient to full-spectrum light, which may be sunlight or artificial light.
Being out in the sun isn't enough by itself, though. We have to soak in the sun's rays. But we've been taught to put on sunscreen whenever we go outside, and there are downsides to the use of sun-blocking chemicals. Our bodies need to be exposed to some full-spectrum sunshine - at least 15 minutes of undiluted sun three times a week [source: USA Today]. The ultraviolet rays of the sun make our bodies produce vitamin D, which is thought to help protect us from various types of cancer and helps us build a store of the vitamin to last through the dark winter months. In addition, vitamin D helps our brains make more serotonin. If we slather our skin with sunblock every time we step out the door, we're cutting down on vitamin D and its benefits.

Benefits of Sunlight: A Bright Spot for Human Health

Each day, Apollo’s fiery chariot makes its way across the sky, bringing life-giving light to the planet. For the ancient Greeks and Romans, Apollo was the god of medicine and healing as well as of sun and light—but Apollo could bring sickness as well as cure. Today’s scientists have come to a similarly dichotomous recognition that exposure to the ultraviolet radiation (UVR) in sunlight has both beneficial and deleterious effects on human health.
Most public health messages of the past century have focused on the hazards of too much sun exposure. UVA radiation (95–97% of the UVR that reaches Earth’s surface) penetrates deeply into the skin, where it can contribute to skin cancer indirectly via generation of DNA-damaging molecules such as hydroxyl and oxygen radicals. Sunburn is caused by too much UVB radiation; this form also leads to direct DNA damage and promotes various skin cancers. Both forms can damage collagen fibers, destroy vitamin A in skin, accelerate aging of the skin, and increase the risk of skin cancers. Excessive sun exposure can also cause cataracts and diseases aggravated by UVR-induced immunosuppression such as reactivation of some latent viruses.
However, excessive UVR exposure accounts for only 0.1% of the total global burden of disease in disability-adjusted life years (DALYs), according to the 2006 World Health Organization (WHO) report The Global Burden of Disease Due to Ultraviolet Radiation. DALYs measure how much a person’s expectancy of healthy life is reduced by premature death or disability caused by disease. Coauthor Robyn Lucas, an epidemiologist at the National Centre for Epidemiology and Population Health in Canberra, Australia, explains that many diseases linked to excessive UVR exposure tend to be relatively benign—apart from malignant melanoma—and occur in older age groups, due mainly to the long lag between exposure and manifestation, the requirement of cumulative exposures, or both. Therefore, when measuring by DALYs, these diseases incur a relatively low disease burden despite their high prevalence.
In contrast, the same WHO report noted that a markedly larger annual disease burden of 3.3 billion DALYs worldwide might result from very low levels of UVR exposure. This burden subsumes major disorders of the musculoskeletal system and possibly an increased risk of various autoimmune diseases and life-threatening cancers.
The best-known benefit of sunlight is its ability to boost the body’s vitamin D supply; most cases of vitamin D deficiency are due to lack of outdoor sun exposure. At least 1,000 different genes governing virtually every tissue in the body are now thought to be regulated by 1,25-dihydroxyvitamin D3 (1,25[OH]D), the active form of the vitamin, including several involved in calcium metabolism and neuromuscular and immune system functioning.
Although most of the health-promoting benefits of sun exposure are thought to occur through vitamin D photosynthesis, there may be other health benefits that have gone largely overlooked in the debate over how much sun is needed for good health [see “Other Sun-Dependent Pathways,” p. A165]. As for what constitutes “excessive” UVR exposure, there is no one-size-fits-all answer, says Lucas: “‘Excessive’ really means inappropriately high for your skin type under a particular level of ambient UVR.”

Vitamin D Production

Unlike other essential vitamins, which must be obtained from food, vitamin D can be synthesized in the skin through a photosynthetic reaction triggered by exposure to UVB radiation. The efficiency of production depends on the number of UVB photons that penetrate the skin, a process that can be curtailed by clothing, excess body fat, sunscreen, and the skin pigment melanin. For most white people, a half-hour in the summer sun in a bathing suit can initiate the release of 50,000 IU (1.25 mg) vitamin D into the circulation within 24 hours of exposure; this same amount of exposure yields 20,000–30,000 IU in tanned individuals and 8,000–10,000 IU in dark-skinned people.
The initial photosynthesis produces vitamin D3, most of which undergoes additional transformations, starting with the production of 25-hydroxyvitamin D (25[OH]D), the major form of vitamin D circulating in the bloodstream and the form that is routinely measured to determine a person’s vitamin D status. Although various cell types within the skin can carry out this transformation locally, the conversion takes place primarily in the liver. Another set of transformations occurs in the kidney and other tissues, forming 1,25(OH)D. This form of the vitamin is actually a hormone, chemically akin to the steroid hormones.
1,25(OH)D accumulates in cell nuclei of the intestine, where it enhances calcium and phosphorus absorption, controlling the flow of calcium into and out of bones to regulate bone-calcium metabolism. Michael Holick, a medical professor and director of the Bone Health Care Clinic at Boston University Medical Center, says, “The primary physiologic function of vitamin D is to maintain serum calcium and phosphorous levels within the normal physiologic range to support most metabolic functions, neuromuscular transmission, and bone mineralization.”
Without sufficient vitamin D, bones will not form properly. In children, this causes rickets, a disease characterized by growth retardation and various skeletal deformities, including the hallmark bowed legs. More recently, there has been a growing appreciation for vitamin D’s impact on bone health in adults. In August 2007, the Agency for Health Care Policy and Research published Effectiveness and Safety of Vitamin D in Relation to Bone Health, a systematic review of 167 studies that found “fair evidence” of an association between circulating 25(OH)D concentrations and either increased bone-mineral density or reduced falls in older people (a result of strengthened muscles as well as strengthened bones). “Low vitamin D levels will precipitate and exacerbate osteoporosis in both men and women and cause the painful bone disease osteomalacia,” says Holick.

Evolution of the Great Solar Debate

In the 2002 book Bone Loss and Osteoporosis in Past Populations: An Anthropological Perspective, Reinhold Vieth, a nutrition professor at the University of Toronto, writes that early primates probably acquired their relatively high vitamin D requirements from frequent grooming and ingestion of oils rich in vitamin D precursors that were secreted by their skin onto their fur. The first humans evolved in equatorial Africa, where the direct angle of sunlight delivers very strong UVR most of the year. The gradual loss of protective fur may have created evolutionary pressure to develop deeply pigmented skin to avoid photodegradation of micronutrients and protect sweat glands from UVR-induced injury.
In the July 2000 issue of the Journal of Human Evolution, California Academy of Sciences anthropologists Nina Jablonski and George Chaplin wrote that because dark skin requires about five to six times more solar exposure than pale skin for equivalent vitamin D photosynthesis, and because the intensity of UVB radiation declines with increasing latitude, one could surmise that skin lightening was an evolutionary adaptation that allowed for optimal survival in low-UVR climes, assuming a traditional diet and outdoor lifestyle. Cooler temperatures in these higher latitudes resulted in the need for more clothing and shelter, further reducing UVR exposure. With shorter winter days and insufficient solar radiation in the UVB wavelengths needed to stimulate vitamin D synthesis, dietary sources such as fatty fish became increasingly important.
Over time, clothing became the norm in higher latitudes and then eventually a social attribute in many societies. By the 1600s, peoples in these regions covered their whole body, even in summertime. Many children who lived in the crowded and polluted industrialized cities of northern Europe developed rickets. By the late 1800s, approximately 90% of all children living in industrialized Europe and North America had some manifestations of the disease, according to estimates based on autopsy studies of the day cited by Holick in the August 2006 Journal of Clinical Investigation and the October 2007 American Journal of Public Health.
Doctors throughout Europe and North America began promoting whole-body sun-bathing to help prevent rickets. It was also recognized that wintertime sunlight in the temperate zone was too feeble to prevent rickets. For this reason, many children were exposed to UVR from a mercury or carbon arc lamp for one hour three times a week, which proved to be an effective preventive measure and treatment.
Around the time the solar solution to rickets gained widespread traction in medical circles, another historic scourge, tuberculosis (TB), was also found to respond to solar intervention. TB patients of all ages were sent to rest in sunny locales and generally returned in good health. Dermatology professor Barbara A. Gilchrest of Boston University School of Medicine says that, whereas sun exposure was shown to improve cutaneous TB, sanatorium patients with pulmonary TB likely responded as much or more to rest and good nutrition than to UVR. Nevertheless, a meta-analysis published in the February 2008 International Journal of Epidemiology found that high vitamin D levels reduce the risk of active TB (i.e., TB showing clinical symptoms) by 32%.
Almost overnight, as awareness of the sun’s power against rickets and TB spread, attitudes toward sun exposure underwent a radical shift. The suntan became valued in the Western world as a new status symbol that signified both health and wealth, as only the affluent could afford to vacation by the sea and play outdoor sports. Phototherapy quickly emerged as a popular medical treatment not only for TB, but also for rheumatic disorders, diabetes, gout, chronic ulcers, and wounds. The “healthy tan” was in, and “sickly-looking” pale skin was out.

Cancer: Cause, Protection, or Both?

The first reports of an association between sun exposure and skin cancer began to surface in dermatology publications in the late nineteenth century. Nevertheless, it was not until the 1930s that the U.S. Public Health Service began issuing warnings about sun-related health risks. People were cautioned to avoid the midday summer sun, cover their heads in direct sunlight, and gradually increase the time of sun exposure from an initial 5–10 minutes per day to minimize the risk of sunburn.
In the decades that followed, the skin cancer hazards of excessive sun exposure would be extensively studied and mapped. Today, the three main forms of skin cancer—melanoma, basal cell carcinoma, and squamous cell carcinoma—are largely attributed to excessive UVR exposure. Skin cancers became the most common form of cancer worldwide, especially among groups such as white residents of Australia and New Zealand.
When atmospheric scientists first called attention to possible chemical destruction of the stratospheric ozone layer in the early 1970s, one predicted consequence of the increased UVB radiation was a rise in skin cancer rates, especially in Australia, New Zealand, South Africa, and Latin America. To counter this threat, the WHO, the United Nations Environment Programme, the World Meteorological Organization, the International Agency for Research on Cancer, and the International Commission on Non-Ionizing Radiation Protection established INTERSUN, the Global UV Project, with the express goal of reducing the burden of UVR-related disease. INTERSUN activities have included the development of an internationally recognized UV Index to help frame sun protection messages related to the daily intensity of UVR. [For more information on these activities, see “WHO Ultraviolet Radiation Website,” p. A157 this issue.]
Australia was among the first countries to spearhead large-scale sun protection programs, with the Slip-Slop-Slap initiative (short for “slip on a shirt, slop on some sun-screen, and slap on a hat”) introduced in the early 1980s. “This program and the subsequent SunSmart campaign have been highly effective in informing Australians of the risks and providing clear, practical instructions as to how to avoid excessive UVR exposure,” says Lucas. As a result of increased use of hats, sunscreen, and shade, the incidence of malignant melanoma has begun to plateau in Australia, New Zealand, Canada, and Northern Europe among some age groups. However, because other UVR-induced skin cancers typically take longer than melanoma to develop, their incidence rates continue to rise in most developed countries. Lucas says a gradual improvement in these rates is to be expected as well.
Whereas skin cancer is associated with too much UVR exposure, other cancers could result from too little. Living at higher latitudes increases the risk of dying from Hodgkin lymphoma, as well as breast, ovarian, colon, pancreatic, prostate, and other cancers, as compared with living at lower latitudes. A randomized clinical trial by Joan Lappe, a medical professor at Creighton University, and colleagues, published in the June 2007 issue of the American Journal of Clinical Nutrition, confirmed that taking 2–4 times the daily dietary reference intake of 200–600 IU vitamin D3 and calcium resulted in a 50–77% reduction in expected incidence rates of all cancers combined over a four-year period in post-menopausal women living in Nebraska.
Moreover, although excessive sun exposure is an established risk factor for cutaneous malignant melanoma, continued high sun exposure was linked with increased survival rates in patients with early-stage melanoma in a study reported by Marianne Berwick, an epidemiology professor at the University of New Mexico, in the February 2005 Journal of the National Cancer Institute. Holick also points out that most melanomas occur on the least sun-exposed areas of the body, and occupational exposure to sunlight actually reduced melanoma risk in a study reported in the June 2003 Journal of Investigative Dermatology.

Other Health Links

Various studies have linked low 25(OH)D levels to diseases other than cancer, raising the possibility that vitamin D insufficiency is contributing to many major illnesses. For example, there is substantial though not definitive evidence that high levels of vitamin D either from diet or from UVR exposure may decrease the risk of developing multiple sclerosis (MS). Populations at higher latitudes have a higher incidence and prevalence of MS; a review in the December 2002 issue of Toxicology by epidemiology professor Anne-Louise Ponsonby and colleagues from The Australian National University revealed that living at a latitude above 37° increased the risk of developing MS throughout life by greater than 100%.
Still to be resolved, however, is the question of what levels of vitamin D are optimal for preventing the disease—and whether the statistical associations reflect different gene pools rather than different levels of 25(OH)D. (Interestingly, Holick reported in the August 1988 issue of The Journal of Clinical Endocrinology & Metabolism that no previtamin D3 formed when human skin was exposed to sunlight on cloudless days in Boston, at 42.2°N, from November through February or in Edmonton, at 52°N, from October through March.)
“Scientific evidence on specific effects of vitamin D in preventing MS or slowing its progression is not sufficient,” says Alberto Ascherio, a nutritional epidemiologist at the Harvard School of Public Health. “Nevertheless, considering the safety of vitamin D even in high doses, there is no clear contraindication, and because vitamin D deficiency is very prevalent, especially among MS patients, taking vitamin D supplements and getting moderate sun exposure is more likely to be beneficial than not.”
As with MS, there appears to be a latitudinal gradient for type 1 diabetes, with a higher incidence at higher latitudes. A Swedish epidemiologic study published in the December 2006 issue of Diabetologia found that sufficient vitamin D status in early life was associated with a lower risk of developing type 1 diabetes. Nonobese mice of a strain predisposed to develop type 1 diabetes showed an 80% reduced risk of developing the disease when they received a daily dietary dose of 1,25(OH)D, according to research published in the June 1994 issue of the same journal. And a Finnish study published 3 November 2001 in The Lancet showed that children who received 2,000 IU vitamin D per day from 1 year of age on had an 80% decreased risk of developing type 1 diabetes later in life, whereas children who were vitamin D deficient had a fourfold increased risk. Researchers are now seeking to understand how much UVR/vitamin D is needed to lower the risk of diabetes and whether this is a factor only in high-risk groups.
There is also a connection with metabolic syndrome, a cluster of conditions that increases one’s risk for type 2 diabetes and cardiovascular disease. A study in the September 2006 issue of Progress in Biophysics and Molecular Biology demonstrated that in young and elderly adults, serum 25(OH)D was inversely correlated with blood glucose concentrations and insulin resistance. Some studies have demonstrated high prevalence of low vitamin D levels in people with type 2 diabetes, although it is not clear whether this is a cause of the disease or an effect of another causative factor—for example, lower levels of physical activity (in this case, outdoor activity in particular).
People living at higher latitudes throughout the world are at higher risk of hypertension, and patients with cardiovascular disease are often found to be deficient in vitamin D, according to research by Harvard Medical School professor Thomas J. Wang and colleagues in the 29 January 2008 issue of Circulation. “Although the exact mechanisms are poorly understood, it is known that 1,25(OH)D is among the most potent hormones for down-regulating the blood pressure hormone renin in the kidneys,” says Holick. “Moreover, there is an inflammatory component to atherosclerosis, and vascular smooth muscle cells have a vitamin D receptor and relax in the presence of 1,25(OH)D, suggesting a multitude of mechanisms by which vitamin D may be cardioprotective.”
To determine the potential link betwen sun exposure and the protective effect in preventing hypertension, Rolfdieter Krause of the Free University of Berlin Department of Natural Medicine and colleagues exposed a group of hypertensive adults to a tanning bed that emitted full-spectrum UVR similar to summer sunlight. Another group of hypertensive adults was exposed to a tanning bed that emitted UVA-only radiation similar to winter sunlight. After three months, those who used the full-spectrum tanning bed had an average 180% increase in their 25(OH)D levels and an average 6 mm Hg decrease in their systolic and diastolic blood pressures, bringing them into the normal range. In constrast, the group that used the UVA-only tanning bed showed no change in either 25(OH)D or blood pressure. These results were published in the 29 August 1998 issue of The Lancet. According to Krause, who currently heads the Heliotherapy Research Group at the Medical University of Berlin, a serum 25(OH)D level of at least 40 ng/mL should be adequate to protect against hypertension and other forms of cardiovascular disease (as well as cancers of the prostate and colon).
William Grant, who directs the Sunlight, Nutrition, and Health Research Center, a research and education organization based in San Francisco, suspects that sun exposure and higher 25(OH)D levels may confer protection against other illnesses such as rheumatoid arthritis (RA), asthma, and infectious diseases. “Vitamin D induces cathelicidin, a polypeptide that effectively combats both bacterial and viral infections,” Grant says. “This mechanism explains much of the seasonality of such viral infections as influenza, bronchitis, and gastroenteritis, and bacterial infections such as tuberculosis and septicemia.” For example, RA is more severe in winter, when 25(OH)D levels tend to be lower, and is also more prevalent in the higher latitudes. In addition, 25(OH)D levels are inversely associated with the clinical status of RA patients, and greater intake of vitamin D has been linked with lower RA risk, as reported in January 2004 in Arthritis & Rheumatism.
Some reports, including an article in the October–December 2007 issue of Acta Medica Indonesiana, indicate that sufficient 1,25(OH)D inhibits induction of disease in RA, collagen-induced arthritis, Lyme arthritis, autoimmune encephalomyelitis, thyroiditis, inflammatory bowel disease, and systemic lupus erythematosus. Nonetheless, interventional data are lacking for most autoimmune disorders and infectious diseases, with the exception of TB.

How Much Is Enough?

Gilchrest points out a problem with the literature: “Everyone recommends something different, depending on the studies with which they are most aligned. One study reports an increased risk of prostate cancer for men with 25(OH)D levels above 90 ng/mL, for example.” In the June 2007 Lappe article, she notes, subjects in the control “high-risk” unsupplemented group had 25(OH)D levels of 71 nmol/L and the supplemented group had levels of 96 nmol/L.
Nevertheless, given the epidemiologic backdrop described above, there are now calls to rethink sun exposure policy or to promote vitamin D supplementation in higher-risk populations. Such groups include pregnant or breastfeeding women (these states draw upon a mother’s own reserves of vitamin D), the elderly, and those who must avoid the sun. Additionally, solely breastfed infants whose mothers were vitamin D deficient during pregnancy have smaller reserves of the nutrient and are at greater risk of developing rickets. Even in the sun-rich environment of the Middle East, insufficient vitamin D is a severe problem among breast-fed infants of women who wear a burqa (a traditional garment that covers the body from head to foot), as reported in the February 2003 Journal of Pediatrics.
Several recent reports indicate an increase in rickets particularly among breastfed black infants, though white babies also are increasingly at risk. A study in the February 2007 Journal of Nutrition concluded that black and white pregnant women and neonates in the northern United States are at high risk of vitamin D insufficiency, even when mothers take prenatal vitamins (which typically provide 100–400 IU vitamin D3). Studies by Bruce Hollis, director of pediatric nutritional sciences at the Medical University of South Carolina, and colleagues suggest that a maternal vitamin D3 intake of 4,000 IU per day is safe and sufficient to ensure adequate vitamin D status for both mother and nursing infant.
These days, most experts define vitamin D deficiency as a serum 25(OH)D level of less than 20 ng/mL. Holick and others assert that levels of 29 ng/mL or lower can be considered to indicate a relative insufficiency of vitamin D. Using this scale and considering various epidemiologic studies, an estimated 1 billion people worldwide have vitamin D deficiency or insufficiency, says Holick, who adds, “According to several studies, some forty to one hundred percent of the U.S. and European elderly men and women still living in the community [that is, not in nursing homes] are vitamin D deficient.” Holick asserts that a large number of infants, children, adolescents, and postmenopausal women also are vitamin D insufficient. “These individuals have no apparent skeletal or calcium metabolism abnormalities but may be at much higher risk of developing various diseases,” Holick says.
In the context of inadequate sunlight or vitamin D insufficiency, some scientists worry that the emphasis on preventing skin cancers tends to obscure the much larger mortality burden posed by more life-threatening cancers such as lung, colon, and breast cancers. Many studies have shown that cancer-related death rates decline as one moves toward the lower latitudes (between 37°N and 37°S), and that the levels of ambient UVR in different municipalities correlate inversely with cancer death rates there. “As you head from north to south, you may find perhaps two or three extra deaths [per hundred thousand people] from skin cancer,” says Vieth. “At the same time, though, you’ll find thirty or forty fewer deaths for the other major cancers. So when you estimate the number of deaths likely to be attributable to UV light or vitamin D, it does is not appear to be the best policy to advise people to simply keep out of the sun just to prevent skin cancer.”
To maximize protection against cancer, Grant recommends raising 25(OH)D levels to between 40 and 60 ng/mL. Research such as that described in Holick’s August 2006 Journal of Clinical Investigation article indicates that simply keeping the serum level above 20 ng/mL could reduce the risk of cancer by as much as 30–50%.
Cedric F. Garland, a medical professor at the University of California, San Diego, says that maintaining a serum level of 55–60 ng/mL may reduce the breast cancer rate in temperate regions by half, and that incidence of many other cancers would be similarly reduced as well. He calls this “the single most important action that could be taken by society to reduce the incidence of cancer in North America and Europe, beyond not smoking.” Moreover, these levels could be readily achieved by consuming no more than 2,000 IU/day of vitamin D3 at a cost of less than $20 per year and, unless there are contraindications to sunlight exposure, spending a few minutes outdoors (3–15 minutes for whites and 15–30 minutes for blacks) when the sun is highest in the sky, with 40% of the skin area exposed.
Holick, Vieth, and many other experts now make a similar daily recommendation: 4,000 IU vitamin D3 without sun exposure or 2,000 IU plus 12–15 minutes of midday sun. They say this level is quite safe except for sun-sensitive individuals or those taking medications that increase photosensitivity.
Gilchrest says some sunlight enters the skin even through a high-SPF sunscreen, so people can maximize their dermal vitamin D production by spending additional time outdoors while wearing protection. “Without the sunscreen, this same individual would be incurring substantially more damage to her skin but not further increasing her vitamin D level,” she says.

Creating a Balanced Message

A growing number of scientists are concerned that efforts to protect the public from excessive UVR exposure may be eclipsing recent research demonstrating the diverse health-promoting benefits of UVR exposure. Some argue that the health benefits of UVB radiation seem to outweigh the adverse effects, and that the risks can be minimized by carefully managing UVR exposure (e.g., by avoiding sunburn), as well as by increasing one’s intake of dietary antioxidants and limiting dietary fat and caloric intake. Antioxidants including polyphenols, apigenin, curcumin, proanthocyanidins, resveratrol, and silymarin have shown promise in laboratory studies in protecting against UVR-induced skin cancer, perhaps through antimutagenic or immune-modulating mechanisms.
Central to the emerging debate is the issue of how to best construct public health messages that highlight the pros and cons of sun exposure in a balanced way. Such messages must necessarily take into account variations in skin pigmentation between groups and these groups’ differing susceptibilities to the dangers and benefits of sun exposure. Moreover, says Patricia Alpert, a nursing professor at the University of Las Vegas, age matters. “The elderly [have a] declining capacity to make vitamin D,” she says. “Many elderly, especially those living in nursing homes, are vitamin D deficient, [even] those living in areas considered to have adequate sunshine.”
Many experts are now recommending a middle-ground approach that focuses on modest sun exposures. Gilchrest says the American Academy of Dermatology and most dermatologists currently suggest sun protection in combination with vitamin D supplementation as a means of minimizing the risk of both skin cancer and internal cancers. Furthermore, brief, repeated exposures are more efficient at producing vitamin D. “Longer sun exposures cause further sun damage to skin and increase the risk of photo-aging and skin cancer, but do not increase vitamin D production,” she explains.
Lucas adds that people should use sun protection when the UV Index is more than 3. As part of Australia’s SunSmart program, “UV Alerts” are announced in newspapers throughout the country whenever the index is forecast to be 3 or higher. “Perhaps,” she says, “this practice should be extended to other nations as well.” U.S. residents can obtain UV Index forecasts through the EPA’s SunWise website (
In the near future, vitamin D and health guidelines regarding sun exposure may need to be revised. But many factors not directly linked to sun protection will also need to be taken into account. “Current observations of widespread vitamin D insufficiency should not be attributed only to sun protection strategies,” says Lucas. “Over the same period there is a trend to an increasingly indoor lifestyle, associated with technological advances such as television, computers, and video games.” She says sun-safe messages remain important—possibly more so than ever before—to protect against the potentially risky high-dose intermittent sun exposure that people who stay indoors may be most likely to incur.

Serotonin, Melatonin, and Daylight

As diurnal creatures, we humans are programmed to be outdoors while the sun is shining and home in bed at night. This is why melatonin is produced during the dark hours and stops upon optic exposure to daylight. This pineal hormone is a key pacesetter for many of the body’s circadian rhythms. It also plays an important role in countering infection, inflammation, cancer, and auto-immunity, according to a review in the May 2006 issue of Current Opinion in Investigational Drugs. Finally, melatonin suppresses UVR-induced skin damage, according to research in the July 2005 issue of Endocrine.
When people are exposed to sunlight or very bright artificial light in the morning, their nocturnal melatonin production occurs sooner, and they enter into sleep more easily at night. Melatonin production also shows a seasonal variation relative to the availability of light, with the hormone produced for a longer period in the winter than in the summer. The melatonin rhythm phase advancement caused by exposure to bright morning light has been effective against insomnia, premenstrual syndrome, and seasonal affective disorder (SAD).
The melatonin precursor, serotonin, is also affected by exposure to daylight. Normally produced during the day, serotonin is only converted to melatonin in darkness. Whereas high melatonin levels correspond to long nights and short days, high serotonin levels in the presence of melatonin reflect short nights and long days (i.e., longer UVR exposure). Moderately high serotonin levels result in more positive moods and a calm yet focused mental outlook. Indeed, SAD has been linked with low serotonin levels during the day as well as with a phase delay in nighttime melatonin production. It was recently found that mammalian skin can produce serotonin and transform it into melatonin, and that many types of skin cells express receptors for both serotonin and melatonin.
With our modern-day penchant for indoor activity and staying up well past dusk, nocturnal melatonin production is typically far from robust. “The light we get from being outside on a summer day can be a thousand times brighter than we’re ever likely to experience indoors,” says melatonin researcher Russel J. Reiter of the University of Texas Health Science Center. “For this reason, it’s important that people who work indoors get outside periodically, and moreover that we all try to sleep in total darkness. This can have a major impact on melatonin rhythms and can result in improvements in mood, energy, and sleep quality.”
For people in jobs in which sunlight exposure is limited, full-spectrum lighting may be helpful. Sunglasses may further limit the eyes’ access to full sunlight, thereby altering melatonin rhythms. Going shades-free in the daylight, even for just 10–15 minutes, could confer significant health benefits.

Other Sun-Dependent Pathways

The sun may be best known for boosting production of vitamin D, but there are many other UVR-mediated effects independent of this pathway.
Direct immune suppression. Exposure to both UVA and UVB radiation can have direct immunosuppressive effects through upregulation of cytokines (TNF-α and IL-10) and increased activity of T regulatory cells that remove self-reactive T cells. These mechanisms may help prevent autoimmune diseases.
Alpha melanocyte-stimulating hormone (α-MSH). Upon exposure to sunshine, melanocytes and keratinocytes in the skin release α-MSH, which has been implicated in immunologic tolerance and suppression of contact hypersensitivity. α-MSH also helps limit oxidative DNA damage resulting from UVR and increases gene repair, thus reducing melanoma risk, as reported 15 May 2005 in Cancer Research.
Calcitonin gene-related peptide (CGRP). Released in response to both UVA and UVB exposure, this potent neuropeptide modulates a number of cytokines and is linked with impaired induction of immunity and the development of immunologic tolerance. According to a report in the September 2007 issue of Photochemistry and Photobiology, mast cells (which mediate hypersensitivity reactions) play a critical role in CGRP-mediated immune suppression. This could help explain sunlight’s efficacy in treating skin disorders such as psoriasis.
Neuropeptide substance P. Along with CGRP, this neuropeptide is released from sensory nerve fibers in the skin following UVR exposure. This results in increased lymphocyte proliferation and chemotaxis (chemically mediated movement) but may also produce local immune suppression.
Endorphins. UVR increases blood levels of natural opiates called endorphins. Melanocytes in human skin express a fully functioning endorphin receptor system, according to the June 2003 Journal of Investigative Dermatology, and a study published 24 November 2005 in Molecular and Cellular Endocrinology suggests that the cutaneous pigmentary system is an important stress-response element of the skin.

Research Challenges

Growing evidence of the beneficial effects of UVR exposure has challenged the sun-protection paradigm that has prevailed for decades. Before a sun-exposure policy change occurs, however, we need to know if there is enough evidence to infer a protective effect of sun exposure against various diseases.
Only through well-designed randomized clinical trials can cause-and-effect relationships be established. However, most sunlight-related epidemiologic research to date has relied on observational data that are subject to considerable bias and confounding. Findings from observational studies are far less rigorous and reliable than those of interventional studies. But interventional studies would need to be very large and carried out over several decades (since most UVR-mediated diseases occur later in life). Moreover, it is not at all clear when, over a lifetime, sun exposure/vitamin D is most important. So for now scientists must rely on the results of well-conducted observational analytic studies.
In sunlight-related research, there are two main exposures of interest: vitamin D status, which is measured by the serum 25(OH)D level; and personal UVR dose, which involves three fundamental factors: ambient UVR (a function of latitude, altitude, atmospheric ozone levels, pollution, and time of year), amount of skin exposed (a function of behavioral, cultural, and clothing practices), and skin pigmentation (with dark skin receiving a smaller effective dose to underlying structures than light skin).
When measuring sun exposure at the individual level, many scientists have relied on latitude or ambient UVR of residence. But these measures are fraught with uncertainties. “While ambient UVR varies, . . . so too do a variety of other possible etiological factors, including diet, exposure to infectious agents, temperature, and possibly even physical activity levels,” says Robyn Lucas, an epidemiologist at Australia’s National Centre for Epidemiology and Population Health. “Additionally, under any level of ambient UVR, the personal UV dose may vary greatly. In short, there is no real specificity for ambient UVR.”
Researchers also assess history of time in the sun at various ages, history of sunburns, dietary and supplemental vitamin D intake, and other proxy measures. Nonetheless, says Lucas, “there are drawbacks to inferring that a relationship with any proxy for the exposure of interest is a relationship with personal UV dose or vitamin D status.” On the bright side, she adds, our ability to accurately gauge an individual’s UV dose history has been enhanced with the use of silicone rubber casts of the back of subjects’ hands. The fine lines recorded by the cast provide an objective measure of cumulative sun damage.

백제의 문화는 일본에 잘 보존되어 있다. comment

고구려나 백제의 문화는 멸망후 반도에는 절멸... 일본으로 넘어가서 오히려 반도보다 그곳에서 더 잘 보존되고 있죠 일본인들의 문화... 야타가라스(삼족오), 사무라이의 갑옷, 도검의 착용법, 승마술, 관복, 상무정신, 예식 등등... 고구려 백제 무사들의 그것과 똑같죠...

90년대는 살기가 좋았다.

엠씨몽님 다른댓글보기
김영삼 시절이전에는
그냥할꺼없으면 공무원누구나할수있는시절이었고
경찰순경도 일반상식수준의 고졸이 지원했고
대학만나와도 삼성 엘지 현대에서 과장부장으로 막스카웃해가던 시절이었지

테니스가 스포츠 중에서 세계 인기 2위고, 개인 스포츠 중에서는 인기 1위다. comment

갤로거   아미르king칸 [zhfldkswha**(고정닉) : 갤로그로 이동합니다.] 조회수178
제 목   테니스 골프가 개인스포츠 투톱아니냐???

전세계에서 가장 인기있는 개인스포츠가 테니스인데 랭킹 100위가 2억벌면 ㄷㄷㄷㄷㄷ

골프는 한국수준에서 놀아도 돈 잘벌던데

2013-01-27 20:26:46

전체댓글수3| 욕설이나 비방 댓글은 누군가에게 큰 상처로 남을 수 있습니다.
테니스 아닐껄|180.224.***.***
  2013.01.27 20:27 
원래 테니스가 고급 스포츠 김치 촌놈들이 골프가 조은지 알지 ㅋ|210.101.***.***
  2013.01.27 20:28 
골프한국수준이 세계수준이다 ㅋㅋㅋ|121.88.***.***
  2013.01.27 20:29 

Saturday, January 26, 2013

말레이시아는 살기 좋은 나라 comment

말레이시아는 빼주세요..
'12.8.18 10:27 AM (
살기 좋은 나라에요. 날씨가 덥긴하지만 나름 계절이 있구요.
우리 나라 여름보단 훨씬 좋아요. 우선 그늘에 있으면 괜챦거든요.
12월에서 1,2월 몬순기간에는 바람이 참 시원하단 생각까지 들어요.
여기 와서 알았어요.
사계절이 좋은 게 아니라는 걸요.
옷이 헤질때까지 입고 버리고..오기전엔 상상이나 했겠어요?

사는 수준도 이슬람은 나라에서 세금,교육,의료,취업등 지원이 많아서
애들도 여럿 낳고 행복지수가 높아요.
우선 기름이 나니 ...외국기업 유치해 세금 왕창 받아 자국민에게 푸는 시스템이지요.
기업에서도 이슬람을 고용해야 하구요.
공무원,경찰,공기업들은 거의 이슬람만 일하구요.요새는 약간 바뀌고 있다지만...
우체국만 가도 모두 이슬람이에요.
대학 갈때도 이슬람 퍼센티지가 있어서 나머지 정원으로 중국계, 인도계들이 치열하게 공부해서 들어가지요.
먹고 살게 해주다 보니 굳이 열심히 살려고는 하지 않죠. 어쨌든 그외 돈되고 좋은 자리는 중국계가 자리잡았네요.
우리가 볼때는 못살아 보일 수 있지만 본인들은 여유롭게 살아요.
성격도 유순하고 되려 중국계나 인도계에 대한 편견이 생겼네요.
살아보니 우리 나라 보다 못산다는 생각보다
우리가 너무 힘들게 살아가고 있구나 절실히 느끼고 있습니다.

Friday, January 25, 2013

우리나라 역사는 경상도 위주

혁신한국이란 자는 소위 보리문둥이라고 통칭되는 경상도 특이족중에서 단순히 특별한 케이스일 뿐이라고 보면 정말 오산입니다. 얘들 우리가남이가족 다수가 암묵적인 합의하에 집단적으로 벌써 이렇게까지 근현대의 정치적 결과를 그대로 과거에 투영한 "만들어진 갱상도 중심역사"의 정교화단계까지 나가고 있습니다. 얘들의 기가막힌, 과학을 빙자한 사이비 소설을 보세요. 나찌의 딱 그것과 일치합니다. 유전학 결과도 딱 지들에게 유리한 일부 결과만 편의적으로 인용하고, 결론을 이미 정해놓고 그런 편취된 자료들을 갖다 붙이기만 할 뿐입니다. 또한, 과거 일본제국주의자들의 멘탈러티, 수법과 아주 유사합니다. 일본족들은 근대화과정에서 서구 백인들에 대한 엄청난 컴플렉스를 갖게 되고, 그에 따라 어떤 식으로든지 죽어라 백인과의 유전적, 문화적 관련성을 강변하기위해 별별 방법을 다 동원합니다. 심지어는 백인들의 조상이 일본이었다는 황당한 주장까지 마치 과학적 주장인 것처럼 유통되고 있습니다. 또 자신의 노골적인 제국주의를 합리화하기 위해 야만적인 사회적 다위니즘을 그대로 차용합니다. 결론적으로, 동서고금을 막론하고 극악한 집단주의가 노정하는 행태는 비슷하게 되어있다는 거죠. 총칼과 부도덕, 비열함으로 더럽게 쌓아올린 자신의 헤게모니를 반드시 정당화해야하기 때문입니다.

경상도족이 내세우는 개소리들의 기본 주장들을 한번 보세요. 징기스칸족과 흉노족의 관련성은 더 따져봐야 될일임에도 불구하고 어쨌든 대충 비슷한 것으로 치부하여 경상도는 이들과 같은 부류이고, 한국역사에서 백제사는 짱꿰사니 일본사니 뭐로 난도질할 수 있어도 고구려사는 남북한 모든 한민족이 부여, 고구려의 영광을 강조할 정도로 막강한 권위를 갖고 있는 이상, 백제사처럼 지들 꼴리는대로 부정하게 되면 지들이 욕쳐먹고 불리할 것이니 아예 "강자"인 부여, 고구려에 억지로 지들의 역사를 줄대는 선택을 하고 있고, 여기서 더 나아가 현재 세계를 지배하고 있는 백인들에게까지 경상도족을 오버랩시키는 쌩코메디를 연출하고 있습니다.

반대로 지들의 개떡같은 궤변의 연장선상에서 "반드시 열등해줘야할" 백제계에 대해서는, 현재의 약자이자, 택도없는 우월의식으로 한국사람들이 얕보는 동남아인들과 지나족에 어떤 식으로든 연관시킬려고 별 쌩쥐랄을 다 떨고 있는 것도 이와 똑같은 맥락입니다.

강원도와 경상도의 부계유전자 구성이 실제로는 큰 차이가 있음에도 걍 닥치고 강원도는 우리 경상도의 형제다란 식으로 씨부리면서 어떻게든 강원도를 자신들의 시다발이로 두려는 것 하며, 김해 대성동 고분은 무슨 부여계니 가야계니 이런 개념보다는 일본 애들이 이를 근거로 임라일본부설을 주장할 정도로 독자적인 색깔의 왜색도 짙은 유적임에도 꼴린대로 지들 유리한대로 빤한 객관적 사실도 난도질하고 있지를 않나, 가야고분에서 나온 유골을 유전자 조사하니 동남아, 그것도 인도, 스리랑카와 아주 흡사했다는 공식적 과학적 조사결과는 아예 쌩까버리고 정반대로 헷소리를 하고 있지 않나, "신성한 피"인 백인의 피를 지들 갱상도족이 가졌다며 자료를 인용해도 지들과 지역적으로 가까운 강원도 지역등의 유적만 예를 들고, 그 보다 더 오래된 북한의 승리산 인골이나 호남의 영산강 유역에서 대규모로 발굴된 전형적인 북구유럽형 유골등에 대해서는 쌩무시를 하고 있지 않나, 신라계가 좋았던 시절은 신라의 당나라에 대한 부역,역적질후 길게봐야 150년이었고 그 이후로 고구려계, 백제계에게 주구장창 철저하게 짓밟히고, 이방원 세력에 의해 갱북 출신의 정도전세력이 척살 당하는 등 조선시대에도 그러한 흐름이 쭈욱 이어져 영조시대에는 반역향으로 찍혀 출사조차 못하는 등 지들의 황제 박정희가 쿠데타로 뒤집을 때까지 장장 1000년이 넘게 천하디 천한 불가촉천민 "보리문둥이"로 불리워온 게 실제 역사적 팩트임에도 해방이후, 친일경상도군사독재 세력에 의해 일방적으로 왜곡된 케케묵은 만들어진 그들만의 역사적 헷소리를 계속 떠벌리고 있지를 않나, 신돌석같은 의병장급이 전라도에는 120명 가까이 됨에도 역사책에도 전부 누락되어있고 독립서훈에서도 대부분 빠져있을 정도(이것은 서울대 사학과 교수의 논문에 나옴)로 해방후 이승만 친일파정권과 경상도군사독재정권의 꼴린대로 정책의 단순한 반영일 뿐인 무슨 본말전도의 극치인 통계자료를 뻔뻔하게 전가의 보도처럼 들이대지를 않나, 그런 문제많은 공식 역사서와 일본, 중국의 사서에서 조차 인정하고 있는 임란 때의 호남의 구국의 항전에 대해서까지 닥치고 헷소리를 하고 있지를 않나, 한 국가내에서 공식적인 제도가 아닌 총칼로, 정경유착으로 억지로 반인륜적 범죄를 저지르면서 쌓아올린 부와 권력도 뛰어난 능력의 징표라고 천연덕스럽게 씨부리지를 않나,,,,

또, 이 전형적인 갱상도족의 헛소리대로 O3가 중국에서 어느 순간 집단적으로 넘어온 지나계이고 이들이 그 전까지 이땅의 주류였던 O2b를 상대적으로 산골짝, 협소한 오지에 불과했던 낙동강전선 동쪽으로 모조리 쑤셔 박아 그곳을 문둥이 집단수용소화할 정도로 막강한 힘과 전투력, 지배력을 가졌고, 그 때이후로 비옥한 한반도 서부를 지배하고, 나라와 왕조를 좌지우지 해 온것이라면, 지금 한반도은 뙤넘말을 쓰고, 뙤넘왕조를 섬기는 지나국의 일개 성이 됐어야 당연히 이치에 맞는 일이 될 것입니다. 과거로 가면 갈수록 압도적인 무력과 지배력을 갖는 지배집단에 의해 언어와 정체성이 그대로 규정되는 것은 철저히 적용되는 보편적인 법칙이기 때문...

그럼에도 이 황당한 갱상도족 개그맨의 정신승리질 보세요. 지나계에 밀려서 소백산맥 밑 문둥이수용소에서 살 수 밖에 없었다고 하면서 또 지들은 신성한 피를 가진 우수한 종족이래요. 이 또라이의 주장에 따르면 인디언 보호구역에 살고 있는 불쌍한 인디언들이 열등한 백인을 압도하는 최고의 우수혈통이 되는 거겠죠. 뙤넘들이 이런 황당한 정신승리질의 대표자인데, 역시 한나라의 노예족이었던 2000년 외노자 갱상도족 답습니다.아 나 이거 참...ㅋㅋㅋㅋ

어쨌든, 다시 말하지만, 이런 황당하고 초딩스런 떼쓰기 궤변을 요런 소수 화상들만 갱상도에서 나불거리고 있다고 착각하면 절대 안된다는 겁니다. 얘들은 남녀노소, 지식인, 무지랭이를 막론하고 거의 무쇠같은 집약력으로 움직이는 유기체적 단일체라는 것을 다른 진짜 한민족이 깨달아야 오늘 날의 개판오분 직전의 나라가 정상화될 거라는 겁니다. 얘들은 이런 류의 종족중의 최악이라고 단연코 얘기할 수 있습니다. 흉노-->한나라노예--->유민도적떼--->천민----->임란 때 7년간 주둔 왜족의 시다발이,,,그러니 얘들이 노골적인 도적떼,해적떼 근성을 유전적으로 보유할 수 밖에 없는 것은 어쩌면 당연한 이치겠죠. 얘들은 얘들답게 대해주면 됩니다. 만약, 전대갈 장군같은 지들의 우상이 서부지역에서 나와 총칼로 싹슬이를 하면 왕, 아니 황제로 추앙할 수 밖에 없는 멘탈리티의 종족이거든요. 얘들에게는 힘과 억지만이 정의이고 최고선입니다.

Ainu people are Greek

great research wahya

just wanted to add that a lot of the words you mentioned have common roots in the ancient hellenic (greek) language

in addition the Ainu decorate their buildings (town hall) and clothing with the ancient symbols of the swastika and hellenic key (as do a numerous ancient buildings/pyramids around the world

as for the timing of the Ainu appearance on the japanese islands you might want to look for evidence (tradition/langage/appearance) of the hellenic diaspora after the altantean/hellenic war resulting in the destruction of the pre-ancient 9000 to 7000 bc hellenic civilation (read SOLON, PLATO) and the sinking of the atlatean peninsula.

I will be posting a more detailed account on the archeological coverups forum

look for signs of the EL (3E)

Arabic language is from Greek

In Middle Eastern and South Asian languages, the common root is "yun" or "ywn". It is borrowed from the Greek name "Ionia", the Greek region of Asia Minor:[6]

    Arabic: يونان (Yūnān)
    Aramaic: ܝܘܢ or יון (Yawān, Yawon)
    Armenian: Հունաստան(Hounastan)
    Azeri: Yunanıstan
    Hindi: यूनान (Yūnān)

    Biblical Hebrew: יָוָן (Yāwān)
        Modern Hebrew: יוון (Yavan)
        KJV Bible: Javan
    Indonesian: Yunani
    Kurdish: Yewnanistan

    Laz: Yonaneti-Xorumona (ხორუმონა)
    Nepalese: यूनान (Yūnān)
    Persian: یونان (Yūnān)
    Sanskrit: यवन(Yavana)
    Tojiki: Юнон (Yunon)
    Turkish: Yunanistan


Thursday, January 24, 2013

조코비치 글루텐 행운 어떤 영양학자가 알려줘

밀가루에 들어있는 글루텐이란 물질에 알러지 반응을 일으키는 증상. 평상시 생활엔 지장이 없지만 운동할때 체력을 조루로 만드는 글루텐 알러지 증상을 세르비아의 한 영양학자가 2010년 말에 조코에게 검사 제안했고 그로 인해 글루텐 프리다이어트 식단으로 바꾸고 나서 체력이 급 호전해서 2011년 무적의 무결점 플레이어로 탈바꿈.|211.208.***.***
정말 운이 좋았네요 역시 나라에 전문가들이 많아서 서로 모르는 거 도와줄 수 있음...운동선수도 우승 할수 있도록 만들어 주고.
2013.01.24 08:42

페이 중국 댄스 상위 5개 엘리트 무용학교 출신 경쟁률 1억대 1임 comment

소년 Talk
highclass (rhdqnr*****) 님 춤배운사람은 알지ㅋㅋ 미쓰에이 안무가 얼만 고난이도인지..
물론 대충 흉내내듯이 추면 쉽지만 미쓰에이처럼 제대로 추려면
다리 성한게 이상한거임 무릎 옆부분 저 정도 상처난거보니
파워있게 다리안무하다 쓸렸구만.. 미쓰에이 민은 미국살때도 유명했음
학교나 유튜브서 코멘트에 동양여자라 안믿긴단 글 달릴정도로 경력 8년
지아,페이 중국 댄스 상위 5개 엘리트 무용학교 출신 경쟁률 1억대 1임ㅋㅋㅋ
경력 둘다 10년 넘어감ㅋㅋ 이런애들이랑 고난이도 아크로바틱 안무에
라이브연습까지 하려니 쉴새없이 연습하니라 수지만 죽어날뿐이지ㅋㅋ
수지도 다른걸그룹안에 들어가면 탑급이지만 경력상 힙합댄스크루 3년출신이니..

Monday, January 21, 2013

North Africans are known as Libyans by the Greek

Berbers, also called Imazighen (in antiquity known as Libyans by the Greek), are the indigenous peoples of North Africa west of the Nile Valley.

Greek and Romans are E1b1b comment

Toronto, Canada

Combining the DNA, the Bible and Roman and Greek Mythology

1) That the E Halopgroup is Hamitic or descendants of Ham (similar to the usage of Jews for Semites using for it the Biblical Character Shem, or similar to the Biblical character Japheth which his descendants now are called Caucasians)

2) I Read a good book called the Faces of the Hamitic People by Khamit Raamah Kush which I found let some light to my Hamitic roots

3) Googled I found that the E-V13 gene pool are Original Roman-Trojan genes (Since Saturn Jupiter and Zeus are associated with Nimrod, a descendant of Cush via Ham, and histories of Nimrod seeking refuge on the 7 hills of Rome and from that point on the Original Romans called him Saturn, Originating the Feast of Saturnilia on December 25)

4) The E-V13 gene root Ancestor was Nimrod the Powerful Hunter Before the Lord also called as well the Orion, Zeus and Jupiter and Saturn etc

5) Ham would be the E Halopgroup ==> Cush (Cronus)= E-M35 ==> Nimrod (Zeus)= E-V13 ==> Dardanus = Dardania ==> Tros = Troy (Trojans)==> Ilus =(Illum) Illyria (Albania)==> Latinus = Latin (the Roman form of the Greek Lateinos) Lateo "to lie hid" or the hidden one)==> Romulos = Founder of the Roman Empire ==> Me an E-V13

6) The Clue for my findings were the Ethiopians and the Yemeni E-M35 as they fit perfectly the Biblical account of Being the descendants of Cush

7) Famous Hamitics E-M35 include Albert Einstein, The wright Brothers, and even Hittler. However looks like throughout history there has been lots of converts of this Hamitic Branch to Judaism which makes 40% of the Jewish Population on the Ashkenazi and Sephardi groups

Roman and Greek are E1b1b comment

Toronto, Canada

No Jensen tell us, which halopgroup you belong and if there is any one as famous as any of the following in our halopgroup e1b1b1? hebrews_dna
Famous E1b1b1 (E-M35)
Albert Einstein (a German-born theoretical physicist who developed the theory of general relativity, effecting a revolution in physics. For this achievement, Einstein is often regarded as the father of modern physics)
Einstein E1b1b1 E-M35.1 M123-, M281-, M293-, M35.1+, M78-, M81-, P72-, V6- m/public/G2c/default.aspx?sect ion=ysnp
Read more:
Shalom ben Amram ben Yitzhaq, 1922-2004 : Samaritan High Priest E1b1b1 Read more:
The Wright Brothers E1b1b1-M35 Y-DNA
were two Americans who are generally credited with inventing and building the world's first successful airplane and making the first controlled, powered and sustained heavier-than-air human. Read more:
William Harvey Famous English physician William Harvey, who was the first in the Western world to describe systemic circulation, belonged to Y-DNA haplogroup E1b1b1.
Borghese Haplogroup E1b1b1 (M35) From the ancient Aramaic "Bar-Ghesse" (Son of Jesse) House of David. Family of Italian noble and papal background. Crypto-Jewish Nobility.
Jewish E1b1b1 haplogroup which accounts for approximately 18% to 20% of Ashkenazi and 8.6% to 30% of Sephardi Y-chromosomes, appears to be one of the major founding lineages of the Jewish population.
Read more:
Conquistadores E1b1b1 haplogroup which accounts for approximately 30% of Latin American Y-chromosomes. Conquistador meaning is "conqueror" in the Spanish and Portuguese languages is the term widely used to refer to the Spanish soldiers, explorers, and adventurers who brought much of the Americas under the control of Spain and Portugal in the 15th to 16th centuries following Europe's discovery of the New World by Christopher Columbus in 1492. The two perhaps most famous conquistadores were Hernán Cortés who conquered the Aztec Empire and Francisco Pizarro who led the conquest of the Incan Empire..
Greek soldiers in Pakistan and Afghanistan
Both E-V13 and J-M12 have also been used in studies seeking to find evidence of a remaining Greek presence in Afghanistan and Pakistan, going back to the time of Alexander the Great. (Y-DNA)
Roman soldiers in Britain
Significant frequencies of E-V13 have also been observed in towns in Wales, England and Scotland. The old trading town of Abergele on the northern coast of Wales (approximately 40%), as reported in Weale et al.(2002). Bird (2007) attributes the overall presence of E-V13 in Great Britain, especially in areas of high frequency, to settlement during the 1st through 4th centuries CE by Roman soldiers from the Balkan peninsula. Bird proposes a connection to the modern region encompassing Kosovo, southern Serbia, northern Macedonia and extreme northwestern Bulgaria (a region corresponding to the Roman province of Moesia Superior), which was identified by Peričic et al.(2005) as harboring the highest frequency worldwide of this sub-clade.[Note 11] (Y-DNA)
Wicked E1b1b1 (E-M35)
Adolf Hitler E1b1b (Y-DNA)
Hitler's father, Alois, a child of a Jewish man called Frankenberger and a German maid, Maria Schickelgruber
Read more:

E1b1b1a is not jews comment

E1b1b1a is not jewish. It is only the sephardic jews who carry E1b1b1a because the sepharidc jews are descendants of berbers who converted to judeaism. The reason why some ashkenazi jews carry E1b1b1a is because they mixed with sephardic jews in western Europe.
People are starting to call all european jews for ashkenazi jews but this is a mistake because the jews from Holland, England, France, Spain and Italy are originally sephardic jews, and the only reason why some ashkenazi jews carry E1b1b1a is because they mixed with the sephardic jews.

The zionists just want to claim E1b1b1a as a ashkenazi jewish DNA so they have a connection to Israel and the medditerrenean area.

And E1b1b1a is carried by many italians and people from balkan, and when Hitlers father was from Austria a country which was colonized by the romans and a neighbor to the balkans where the majority carry E1b1b1a then it is not strange that Hitler carried E1b1b1a.

Portugal E1b1b 12 percent=E-M81+other subclades

vrkas fieldvanreest 7 months ago

12% in Portugal is the total of all subclades of E1b1b. E-M81 is just one subclade. E-M81 (6%) + other subclades = 12 % E1b1b in Portugal.

R1b is from africa


E(V13) is not Middle Eastern. Neither is E in General it is African and European.
If E(V13) is not European R1b is African and Central Asian!
Hitler has a typical gene for Balkan and Central Europe! Descendant from one of the first Europeans!
Typical for Jews are E(V22),J1 and some subclades of J2 ,R1a and R1b

E1b1b paternel ancestor still lived for sure in Africa "only" 18 000 year ago, that is 40 000 years after the "out of africa" migration comment

Antoine1706 said...
"Dienekes is exactly correct in that an identification of E1b1b is nearly useless in identifying Hitler's paternal lineage and background. It would not be at all surprising to discover that he was V13, since the Waldviertel borders on the Danube and that is a well-known migrational corridor out of the Balkans to the remainder of Europe. Other origins are less likely. (I have serious doubts that Hitler's family was Somalian in origin, for example.)"

It does not matter the subclades E-M78,E-V13, E-M81 etc he belonged to. By the way even E-V13 is still closer to any other E1b1b subclades than to R1b or I for example...

What is important is that his E1b1b paternel ancestor still lived for sure in Africa "only" 18 000 year ago, that is 40 000 years after the "out of africa" migration ! So even if his autosomal profile was without any doubt fully european, claiming an "Aryan" ancestry with such an haplogroup would have been nevertheless been difficult under the nazi rule...

Ancient Romans and Greeks belong to E1b1b group that hitler might inherit it from comment

Dean said...
If Hitler did in fact partially descend from ancient Greeks (via Latins who were mixed with Greeks from the Roman era or who were Latinised Greeks?), how ironic would it be in that he and subsequent Nordic supremacists believed that ancient Greeks, and/or Spartans in particular, were descended from ancient "Nordic" blond people, when the reverse is more plausible, even in low numbers among modern Germans?

There is a comedic element in that the hero of the white supremacists has been officially knocked off of his pedestal.

Sunday, January 20, 2013

뇌 신경세포는 밤낮이 바뀌는 것을 극도로 싫어한다.

뇌 신경세포는 밤낮이 바뀌는 것을 극도로 싫어한다. 해가 지면 멜라토닌이라는 호르몬을 분비해 잠을 잘 수 있도록 하고 햇빛을 쬐면 세로토닌이라는 호르몬을 생산하는 것이 뇌신경세포의 역할이다. 이것을 인간이 뒤바꾸면 뇌신경세포에 혼란을 가져오게 된다. 이처럼 햇빛의 유무에 따라 뇌 속에 뇌효율을 결정하는 호르몬이 달라진다.

특히 햇빛을 쬘 때 뇌신경세포가 만들어내는 세로토닌이라는 호르몬은 마음의 안정을 가져다주는 역할을 한다. 불안감에 시달리기 쉬운 입시생은 반드시 적당한 햇빛이 필요한 것이다. 그러므로 공부를 하되 반드시 낮에, 그것도 햇빛이 잘 드는 방에서 가능하면 창가에 앉아 하는 것이 가장 좋다. 낮에 커튼을 친다든가, 밤에 전기불 아래서 공부하는 것 역시 정상적인 호르몬 분비를 방해한다. 세로토닌은 전기불에는 절대로 반응하지 않기 때문이다.

콩나물을 예로 들어보자. 콩을 땅에 심어 햇빛과 물, 거름, 토양 등 자연의 조건을 갖춰주면 각각의 유전자가 활발하게 움직여 잎사귀와 줄기, 꽃을 피워내지만 햇빛과 토양이 없는 곳에서 물만으로 키운 콩나물은 잎사귀가 될 유전자와 꽃이 될 유전자가 죽어버린다. 입시생들이 햇빛을 쬐지 않고 자연을 접할 기회를 전혀 갖지 못하게 되면 비정상적인 콩나물처럼 되는 것이다.

또 햇빛이 피부에 닿아야 피부 속의 콜레스테롤을 비타민D로 바꿀 수 있으므로 혈당을 떨어뜨려 혈압도 낮출 수 있다. 스트레스가 쌓일 때 밖으로 나가 햇빛이 드는 잔디밭을 바라보고 새소리를 듣고 있으면 저절로 마음이 가라앉는 것을 느낄 수 있다. 눈부신 햇빛, 넓은 바다, 푸른 하늘 등 자연환경이 뇌속의 세로토닌과 엔돌핀 등 중요한 호르몬을 자극하는 것이다. 공부에 바빠 밖으로 나갈 시간이 없다면 최소한 방안에 햇빛이 잘 들게 하고 자연을 담은 사진이나 그림이라도 걸어두면 좋다. 답답한 마음이 들 때마다 벽에 걸린 경치를 바라보며 심호흡을 하면 한결 기분이 가벼워진다.

Saturday, January 19, 2013

Hebrew is Greek comment

You're hilarious and uneducated just because you don't wanna go with the worlds recognized history, your word has NO proof because it's just an stupid idiotic opinion and nothing more! and about the Jews you should know something, you can fnd Jews that said HEBREW IS GREEK, but you vever will find a Hellene that said Hellenic is Hebrew! you are really funny to chat with ha ha look up (Youtube) HEBREW IS GREEK- and you can find the Jews that wrore all about there real origin! CHEERS
· 응답 대상: NoSirNoSerbs (댓글 표시) 

Greece means greatness comment

i already know that you're really stupid, but the good thing here is that the whole world gets to read your stupid comments! the name Greece comes from the word grecia, that originates from the Italian language and it means greatness, (country of the great ones), even before the Romans Greece was called Hellas and until today you ignorant muppet, your name originates from the Caspian Albania and you can't do anything about the reality!
· 응답 대상: NoSirNoSerbs (댓글 표시) 

Italians came from greek comment

Latin originates from the ancient Italic world you muppet berisa,) and the mother language of Latin is the ancient Hellenic, you can ask this to anybody from Italy you ignorant wannabe!
· 응답 대상: NoSirNoSerbs (댓글 표시)
go on and ask any Italian where does his language come from?? ha ha you stupid muppet ha ha ha!! and don't make me talk about your mother, she was working really hard in Athens srteets so you can live in a house over there,
· 응답 대상: NoSirNoSerbs (댓글 표시)

한국 좌파 신문의 유럽 국가들 얕보기

[한겨레] 미야지마 히로시, 나의 한국사 공부

미야지마 히로시 지음/너머북스·2만3000원

“일 본 봉건제론은 일본이 러일전쟁에 승리하면서 이른바 세계적 열강의 일원으로 등장했던 시기에 일본과 유럽의 역사적 동질성을 주장하기 위해 ‘발견’된 것으로서, 처음부터 이데올로기적인 성격이 강한 역사인식이었고 일본사 연구의 탈아(脫亞)적인 경향을 보여주는 주장이었다. …일본 봉건제론은 근본적으로 재검토되어야 한다.”

이는 2002년부터 성균관대 동아시아학술원에서 연구와 강의를 해온 일본 역사학자 미야지마 히로시(65·사진) 교수의 주장이다. 미야지마 교수는 교토대에서 일제시기 조선 노동운동, 조선 농촌경제 변동, 토지조사사업 연구로 학위를 받고 도카이대와 도쿄도립대, 도쿄대 동양문화연구소 교수를 거쳐 성균관대로 온 한국사 및 동아시아사 연구(경제사)의 독보적인 권위자다. 그의 주장대로라면, 일본엔 유럽식 봉건제가 존재한 적이 없다. 그럼에도 일본 주류 역사학자들이 그렇게 주장한 건 결국 제국주의 식민사관 논리를 뒷받침하기 위해서였다.

한국·동아시아 경제사 연구 권위자

“유럽 중심 사관을 기준으로 삼아

동아시아 역사 규정하는 것은 잘못”

미야지마 교수는 근대 이전의 일본이 신분제 사회였다는 점에서는 조선·중국과 확실히 달랐다고 본다. 다이묘나 하타모토 같은 상층 무사계급은 장자 가문(이에) 중심으로 통치 지위를 세습했고, 농업·상업·수공업을 가업으로 삼는 이에들도 존재했다. 하지만 조선·중국은 신분제 사회가 아니었다. 중국은 송·명대 이후 중앙집권적 관료체제가 확립된 뒤로 오히려 신분제는 쇠퇴했다. 지배그룹은 과거에 합격한 개인들을 중심으로 형성됐으며, 그들의 지위는 세습되지 않았다. 이를 받아들인 조선도 중국보다는 다소 폐쇄적인 지배 그룹이 형성되긴 했지만 신분제는 쇠퇴했다. 양반은 세습적·고정적 신분이 아니었다. “지배 신분에 속하는 자가 지역주민의 70~80%나 되는 현상은 신분제 국가에서는 있을 수 없는 일이다.”

이런 사실들을 입증하기 위해 토지소유와 국가체제, 신분제, 지배계층, 가족과 친족 등을 이웃 나라들의 그것과 비교하며 다각적으로 살피는 이 일본인 연구자의 평생을 기울인 노력은 치밀하고 신선하다.

일본에선 왜 신분제가 존재했을까? 가장 큰 원인은 시장경제가 발달하지 못했기 때문이라고 그는 설명한다. 신분제는 사회적 분업이 자동적으로 이뤄지는 시장이 없을 경우 지배층이 이를 강제하기 위한 장치다.

전근대 유럽이 신분제 사회였던 이유도 시장이 발달하지 못했기 때문이다. 10세기께부터 시장이 등장한 중국은 18세기까지, 적어도 16세기까지는 세계에서 가장 선진적인 사회였다. 송나라의 신유학(송학)과 그것을 집대성한 주자학, 주자학이 채점기준이 된 과거제와 실력 위주의 관료등용제 확립, 신분제의 해체, 농업과 상공업의 비약적인 발전과 상품·화폐 경제의 진전이 그것을 대표한다. 그런 상황과 조응하는 사회구조를 미야지마 교수는 ‘소농사회’라고 부른다. 소농사회는 자신의 토지를 소유하거나 다른 사람 땅을 빌리거나 간에 기본적으로 자신과 그 가족의 노동력만으로 독립적인 농업경영을 하는, 소농의 존재가 지배적인 농업사회를 가리킨다. 이는 대토지소유제를 토대로 한 유럽 영주계층의 직영지 경영 방식이나 광범위한 농업노동자들이 존재한 동남·서남아시아와는 근본적으로 다른 점이다. 이런 독특한 소농사회가 중국에서는 송·명대 이후, 한국·일본에선 각기 양상이 좀 다르긴 하지만 17세기 이후 성립됐다.

이에 비하면 같은 시기의 유럽은 훨씬 후진적인 사회였다. “그럼에도 불구하고 유럽 봉건사회를 기준으로 중국사, 동아시아 역사를 규정하는 것은 큰 잘못이 아닐 수 없다.” 따라서 고전고대·중세봉건·근대 부르주아라는 단선적인 유럽적 발전사관을 표준으로 삼는 기존 역사인식이나 시대구분은 잘못됐다는 것이다.

미야지마 교수는 1991년에 자신이 <조선 토지조사사업사의 연구>라는 첫 단행본을 냈을 때 한국 사학계 일부가 일제를 미화했다며 비판한 것도 유럽중심 사관과 연관이 있다고 본다. 조선도 일본처럼 자본주의를 향한 자생적·내재적 발전과정을 거쳐왔는데, 일제가 오히려 이를 망가뜨렸다고 보는 ‘자본주의 맹아론’이 그중의 하나다. 식민지근대화론을 주장하는 뉴라이트 계열 연구자들이 자본주의 맹아론을 비판했고, 미야지마 교수가 그들의 연구에 관심을 보인 것도 그에 대한 오해를 부추긴 것으로 보인다.

하지만 동아시아 소농사회가 오히려 유럽보다 앞섰다고 보는 미야지마 교수에게 자본주의 맹아론 등은 유럽중심 사관에의 지나친 함몰이요 견강부회일 수 있다. 그는 그런 관점에서 한국 역사교과서들도 조목조목 비판한다.

내년 2월 정년퇴임을 앞둔 미야지마 교수는 이 서구중심적인 역사인식 패러다임을 뛰어넘어 한국·동아시아에 맞는 새로운 학문방법론을 수립하는 걸 여생의 주요 과제로 삼고 있는 듯하다. 한승동 기자

중세 유럽 나라들을 내리 까는 전형적인 한국 동양인의 거짓말

전근대 유럽이 신분제 사회였던 이유도 시장이 발달하지 못했기 때문이다. 10세기께부터 시장이 등장한 중국은 18세기까지, 적어도 16세기까지는 세계에서 가장 선진적인 사회였 다. 송나라의 신유학(송학)과 그것을 집대성한 주자학, 주자학이 채점기준이 된 과거제와 실력 위주의 관료등용제 확립, 신분제의 해체, 농업과 상공업의 비약적인 발전과 상품·화폐 경제의 진전이 그것을 대표한다. 그런 상황과 조응하는 사회구조를 미야지마 교수는 ‘소농사회’라고 부른다. 소농사회는 자신의 토지를 소유하거나 다른 사람 땅을 빌리거나 간에 기본적으로 자신과 그 가족의 노동력만으로 독립적인 농업경영을 하는, 소농의 존재가 지배적인 농업사회를 가리킨다. 이는 대토지소유제를 토대로 한 유럽 영주계층의 직영지 경영 방식이나 광범위한 농업노동자들이 존재한 동남·서남아시아와는 근본적으로 다른 점이다. 이런 독특한 소농사회가 중국에서는 송·명대 이후, 한국·일본에선 각기 양상이 좀 다르긴 하지만 17세기 이후 성립됐다. 

이에 비하면 같은 시기의 유럽은 훨씬 후진적인 사회였 다. “그럼에도 불구하고 유럽 봉건사회를 기준으로 중국사, 동아시아 역사를 규정하는 것은 큰 잘못이 아닐 수 없다.” 따라서 고전고대·중세봉건·근대 부르주아라는 단선적인 유럽적 발전사관을 표준으로 삼는 기존 역사인식이나 시대구분은 잘못됐다는 것이다.

중국은 한족의 나라다

현대 중국의 창시자 쑨원은 만주족의 국가 청을 무너뜨리고 중화민국을 수립한 후 내각을 이끌고 명나라 영락제의 묘를 찾아갔다.
1644년 만주족이 명을 정복한 이후 중국인들이 세운 국가의 첫 중국인 지도자로서 쑨원은 조상들에게 인사를 올리러 간 것이다.
유교식으로 제물을 바친 그는 조상들이 아직 살아 있는 듯 이렇게 이야기했다.

" 만주족의 정책은 ... 지독한 학정이었습니다. ... 영원한 한족 종속의 욕망에 의해 ... 만주족의 지배로 나라는 영구적인 피해를 입었고 백성들은 고통을 받았습니다. ... 오늘의 한족은 ... 마침내 한족의 정부를 재건했습니다. ... 조상님들의 후손들이 오늘 이 자리에 승리를 알려드리려고 왔습니다."
(1912년에 출판된 Herbert A. Giles의 저서 Chapter 12 중)

원뿐만이 아니라 중국 전역의 한족들이 1911년 혁명 시기에 변발을 자름으로써 만주족의 지배에서 벗어났다는 것을 보여주었다.
중 국의 붉은 별Red Star over china이라는 에드거 스노Edgar Snow의 책을 보면 마오쩌둥은 한 전투가 끝나고 '한漢깃발이 게양되는 것'을 보았으며, '한'이란 글자가 들어간 하얀 깃발이었다.'고 말했다. 한은 한족을 가리키는 말이다. 혁명은 중국인이라는 뜻의 단 한 글자를 단 깃발을 날린 것이다.
19세기에 일어난 태평천국운동 때 중국인들이 수천 개의 깃발 아래서 만주족 수천 명을 학살하는 것을 보았기 때문에 깃발만으로도 만주족들은 공포에 휩싸였다.

1911 년부터 중화인민공화국이 수립된 1949년까지 내내 만주족들은 중국 땅에서 살기 위해 민족 정체성을 숨겼고, 집 안에서만 몰래 아이들에게 모국어를 가르쳤다. 1911년 중국 곳곳에서 만주족 제거 움직임이 일어나 수천에 달하는 만주족들이 중원을 탈출해 만주로 돌아갔다.

전문직을 가지신후 머리를 굴리면서 편하게 일하던지

답은 간단함 남자라면
대학을 가신후 전문직을 가지신후 머리를 굴리면서 편하게 일하던지 그런후 나중에 경험 좀 쌓이면 자기 비지니스를 하는게 도움이 됨
아님 확실한 기술을 소유하고 계시던지 역시 이것도 나중엔 비지니스로 연결됨

여자라면 뭐 아무 능력없어도 돈 벌수있는 건 정말 많음.......
무슨말인지 잘 알것으로 생각함
현실이 이런걸 뭐.......

Wednesday, January 16, 2013

French is beautiful language

Hi everybody!

Even if I am not french, french is my mother tongue , I also speak arabic, spanish and english and I would say, witouh ANY DOUBT, that french is the most beautiful language to say romantic things, and as Celine Dion already said, just before singing s'il suffisait d'aimer: "le français est la plus belle langue pour dire des mots d'amour".
I read arabic, french, spanish and english poetry but when I read it in french, every single word sounds as if it was sung...
I also listen to all kind of musics, even indian music, but when it's in french it makes me quiver.... how can you compare a pour que tu m'aimes encore with a if that´s what it takes. where can you find another Brel or another Aznavour or another Verlaine, Hugo, Musset...?
However, I do love the other languages I speak and I do love English language!

France military tech is 30 years ahead of the US

You saw what we did to Iraq in 1991. France's military tech is 30 years ahead of what we had then, and the Arabs for the most part are still using technology from then. And don't forget the Arabs training is so bad it isn't even funny, as where French training is some of the best in the world.
· 응답 대상: TheVanezi (댓글 표시) 

Islam gave blacks freedom arrogance comment

you fucking niger, read the story, you were slavery before Mohamed (sala allah alayhi wa salam) ,and islam give your fucking ass freedom.....but you still denied who you are and where you come from, and at this point i can understand the darwin's theory....
· 응답 대상: king africa

Saturday, January 12, 2013

Ancient Greeks were full Mediterranean

In Ancient times the Greeks were dark skinned peoples, this is supported by many historical evidences, Herodotus (a Greek historian writer) says that the Persian troops who were in greece were pale compared to the tanned skinned greek troops. another evidence about him saying about the skythians who were fair featured with red hair passing through greece, he says they came through Asia minor into greece.
· 응답 대상: yashil17 (댓글 표시) 

Italians are from Greek

  • Sam Angelo
    Saxons ARE Germanic. So are the french and Spanish. All of the northern Europeans like German, English and even all Scandinavian people originated Spain and France. Known as Iberia or Basque lands. Italians are the indo-european(east europe; Turkey, Greece, Romania) mixed with a bit of the Iberian basque that had migrated down to North Italy. Real Italians are originally dark.
    · 응답 대상: Psythr666 (댓글 표시)
    • Sam Angelo
      Yes. I know italian ARE indo-european. I know the celts ARE Basque. It is exactly what I said. But Iberia is in Spain. NOT Italy. I know the Northern Italians were basque: white. But they were never celts. Italians in Early Roman times were Indo-European(Dark). The Roman Empire was after the Basque had gotten into Northern Europe. During the Roman lifetime, some Northern Europeans traveled south into Italia. But not Celts.
      · 응답 대상: Joe Mills (댓글 표시) 
    • Sam Angelo
      NO!!! Europeans are NOT Greek!!! REAL Europeans are directly from North Africa!! They can be classified as Iberians since the first piece of European land they touched was Spain. Then they traveled up all the way to Scandinavia. THOSE are Europeans!! Not Greeks!! 
    • Sam Angelo
      YOU LOOK UP YOUR HISTORY!!! Unless your history IS of Greek descent than good for YOU!!! But There were already people in Europe before the Greeks even had their language!!! ROMANS??!! I LAUGH SO HARD!!! The romans WERE Greeks!!! But they go back 2 thousand years ago. There were already people in Germany THOUSANDS of years before the fucking Greeks left East Asia!!! WHITES ARE FROM AFRICA DIRECTLY!!!! Greeks didn't even exist yet!!!!
      • Sam Angelo
        No you fucking moron. Don't fucking comment to me again on how I'm wrong. The Celt came from Iberia. There was once discovery that the DNA of the Celtic nations like Ireland have a same code as the Iberians. They traced their DNA all the way to Spain. People in Iberia actually have more of this piece of the celtic DNA than the people of the Celtic lands. They found out that the Celtic people had migrated from Iberia. And in 2007 it was finally proven that ALL of Europeans arrived from Iberia.
        • Sam Angelo
          P1: I was wrong to say the first Europeans touched Iberia. That was incorrect. The first known Europeans were in Georgia. But not all Europeans are the same. The people that were in Georgia 1.8 million years ago are related to our Greeks of today. The people now of Asian Minor like Greece and Turkey are descendants of these first Europeans of Georgia. BUT there are other kinds of Europeans. Ones that DID arrive not from East Africa to Asia minor, but from the West into Iberia.
        • Sam Angelo
          P2: They had arrived 1 million years ago in Iberia directly as Africans. And throughout centuries they populated all West Europe. These people were descendants of Neanderthals, not Homo-Georgicus like the first Europeans in Georgia. They had arrived .8 billion years after the Georgians in South East Europe. The Georgians had a headstart and were more civilized than the West Europeans. East Europeans hatched Mesopotamia, Greece and the Romans were also of Origins to the Georgians.
        • But the people of the Balkans are not the same as the Europeans of the west and Northwest you fool. The people of the 'Balkans' got to Europe first. Then years after another group of Euros traveled from the piece of Africa that is right below Spain. I HAD TO EXPLAIN MYSELF ALL OVER AGAIN FOR YOU!! DIDN'T YOU PAY ATTENTION??!!
          · 응답 대상: amasonga (댓글 표시)
          P3: People from Scandinavia down to Portugal ARE related to each other. The most profound and recent(2007) evidence of the people with origins to Iberia are of the people in the Isles around England. They detected a total similarity to the people in Iberia today. Skull structures were the same. As well as the people of the Celtic Nations. They traced a code in people of the Celtic lands all the way to Spain and found out people in Iberia have more of this celtic patch in their DNA
          · 응답 대상: Nata Mikashavidze (댓글 표시) 


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